Legacy biology claims that aggressively reproducing bacteria are responsible for cell death in the lung tissue. The body tries to frantically repair the damage whilst the immune system is responsible for killing off the bacteria at the same time.
The New Biology paradigm is happier with the idea that it is the tissue that dies first and that the bacteria are not causal in the process but are merely opportunist scavengers that live off dead tissue.
But what causes the tissue necrosis in the first place and why is the lung tissue seemingly more susceptible to this type of disorder than other parts of the body? Why is pneumonia common in hospitals with supposedly strict hygiene protocols and why does it seem to be a progression of other respiratory conditions such as influenza. Why don’t the nurses ‘catch’ it?
First consider that the lung tissue needs a continuous supply of energy in order to maintain it. This is assumed to come from oxygen in the blood delivered via the capillary system. The job of the lungs though is to absorb oxygen from the lung cavities and deliver it to the rest of the body and this is achieved via a separate capillary system, the pulmonary capillaries.
The coexistence of two such systems is a complexity not seen in the rest of the body and I will guess that this restricts the number of maintenance capillaries somewhat thereby making the whole system a little delicate and meaning that any extra input of energy in this area would be most welcome.
Konstantin Meyl has stated that such an additional input exists in the form of electromagnetic field vortices which are transferred from fresh air through the lung tissue directly to the bloodstream. Air that has been breathed and had insufficient time to recover is depleted of vortices and depleted of energy.
Gerald Pollack has written a paper going a step further, claiming that there is no exchange of oxygen at all in the lungs and that all energy input is via electrical energy.
Hypothesis: This energy is not merely necessary as an input to the bloodstream, but is vital for the maintenance of local lung tissue. These vortices will be absorbed directly into the lining of the lungs and assist in maintaining healthy cells. Exercise will increase breathing and proportionately increase energy intake. The inhalation of stale air will reduce energy intake.
We can see now the possibility of necrosis prior to bacterial proliferation.
An already weakened patient is confined to bed and immediately suffers a decrease in energy input to the lung tissue and in due course the intake of stale air further reduces available vortex energy.
Nurses and carers do not succumb as they are walking around, breathing more air and not spending 24 hours a day inhaling ‘dead’ gases.
The disease seems to be a progression of a viral infection but it is a consequence of bad treatment instead.
Treatment
If the cause is a lack of energy in the air then we should expect that the treatment should consist of .. exposure to fresh air!
The open air treatment of pneumonia – W P Northrup
https://jamanetwork.com/journals/jama/article-abstract/460480
“Our systematic practice was to put all pneumonia patients during the day, for six hours, on the roof, in the open air, in all weather in which harsh high winds, rain and snow did not prohibit. Indeed, the patients were not always brought in for little sprinkling rains or trivial snowfalls, and many times were out when high snow banks formed a corral about the space in which the beds were grouped” – Northrup (1906)
“Gradually, after most careful precautions and constant watching, it became the firm conviction of all observers that such patients were decidedly benefitted thereby.”
References:
Is it oxygen, or electrons, that our respiratory system delivers? – Gerald Pollack
https://www.sciencedirect.com/science/article/abs/pii/S030698772400210X
The open air treatment of pneumonia – W P Northrup
https://jamanetwork.com/journals/jama/article-abstract/460480